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Additional resources for Anaesthesia, Pain, Intensive Care and Emergency Medicine — A.P.I.C.E.: Proceedings of the 10th Postgraduate Course in Critical Care Medicine Trieste, Italy — November 13–19, 1995
From the brain to the mind We can say that pain is a complex mental phenomenon evoked by nociception (29). If the areas of our brain cannot be activated and become refractory to receive neuronal communication, the transition from nociception and pain does not occur. Nociception remains blocked in the thalamus and is not received by the cortex. This happens under general anesthesia or when a person loses conSCIOusness. There are particular mental states, such as a major distraction caused by events that are of great interest to the individual (the classical example of the soldier in battle) that can alter the level of algic threshold up to analgesia.
Changeux J-P, Connes A (1989) Matiere a Pensee (ed) Odile Jacob, Paris 31. Eccles J (1991) I meccanismi della percezione del dolore: dai recettori sensoriali, alla corteccia cerebrale e da questa alIa mente. Seminari suI dolore, 2:5-26 32. Eccles J (1994) Come rio controlla il suo cervello. Rizzoli, Milano 33. Crile GW (1910) Anoci-association. Saunders, Philadelphia 34. Bonica JJ (953) The management of pain. Lea and Febiger, Philadelphia, pp 1240-244 35. Morpurgo CV, Spinelli DN (1976) Plasticity of pain perception.
Exploratory laparotomy was carried out in these animals during continuous brain stimulation without the use of chemical anesthetics. Reynolds concluded that focal brain stimulation in this region can Acute Pain Modulation in the Central Nervous System: A Chronological Review 47 induce analgesia in the absence of diffusely applied "whole brain" stimulation (17). David Mayer, a collaborator of John Liebeskind of the University of California at Los Angeles, heard one of Reynolds' presentations and, with Liebeskind and Akil, repeated and expanded this line of research (18).